What's Behind Boils and Abscesses?

REVIEWED BY

William Maish, MD MBA MPH

Clinical Product Lead

Published

Last updated

Quick answer:

A boil is an abscess of a single hair follicle, while an abscess is any walled-off pocket of pus in the skin. Staphylococcus aureus causes most of them, isolated in 76% of purulent skin infections in one US emergency department study. Drainage, not antibiotics, is the definitive step: 68.9% of drained abscesses 5 cm or smaller had resolved at 10 days on placebo alone, and about 10% of people have another within 12 months.

Read more →

See the markers behind recurrent skin infections

Check the metabolic and inflammatory markers behind repeat skin infections.

  • CLIA-certified labs
  • HIPAA compliant
  • Personalized protocols
Book your test

Boils vs. abscesses: what's the difference?

A boil, also called a furuncle, is an infection of a single hair follicle in which pus extends through the dermis into the fatty tissue beneath, forming a small abscess. A skin abscess is the broader term for any walled-off pocket of pus in the skin, usually felt as a tender, fluctuant red lump. Every boil is a kind of abscess.

That distinction matters more than it sounds, because the two words get used interchangeably in clinics and by search engines alike. Here is how the whole family of lumps sorts out.

LesionHow deep it sitsTypical sizeWhat causes itWhere it usually appearsDrains on its ownUsual management
Boil (furuncle)Follicle down into the subcutaneous fatPea to grapeBacterial infection of one hair follicleFace, neck, armpits, buttocks, thighsOften, once it comes to a headWarmth and cleanliness; drainage if large
Skin abscessDermis and deeper skin tissuesGrape to golf ball or largerBacteria walled off into a pus cavityAnywhere on the body, skin or deeper tissueSometimes, unreliablyIncision and drainage
CarbuncleSeveral linked follicles into deep subcutaneous tissueLarger than a single boilTwo or more contiguous furunclesBack of the neck, back, thighsPartially, via multiple draining sinus tractsIncision and drainage
Epidermoid cystSac sitting within the skinPea to walnutTrapped keratin, not infectionFace, neck, trunk, backNo, unless it ruptures or becomes infectedLeft alone, or removed if it inflames
CellulitisDeep dermis and subcutaneous tissueSpreading area, not a lumpBacteria spreading diffusely through tissueLegs most often, but anywhereNo, there is no drainable pocketAntibiotics, no drainage

Boil (furuncle)

A boil starts where a hair does. Bacteria settle in a follicle, the follicle inflames, and the infection pushes downward. That downward push is what separates a furuncle from folliculitis, where inflammation stays more superficial and pus is limited to the epidermis. Same organism, same starting point, different depth. If a clinician writes "furuncle" on your notes, you have a boil.

Skin abscess

An abscess is what you can feel: a collection of pus within the dermis and deeper skin tissues, presenting as a painful, tender, red nodule that gives slightly under a fingertip, the way a grape does compared with a marble. That springy quality has a clinical name, fluctuance, and it is the sign that liquid pus has collected rather than simply inflamed tissue. Abscesses are not confined to skin either; the same walled-off pocket can form in a tooth, an organ, or deeper soft tissue.

Carbuncle, cyst, and cellulitis: where they fit

These lesions sit on a single line rather than in separate boxes. Folliculitis can deepen into a furuncle, and when several infected follicles run together the result is a carbuncle, an infection that extends into surrounding skin and deep subcutaneous tissue with multiple draining openings, most often on the posterior neck, back, and thighs where skin is thicker. Two things get mistaken for that progression. Cellulitis spreads outward as a flat, expanding red patch with no pocket to drain. And a cyst is a non-infected sac that sits quietly for years until it inflames, at which point it mimics a boil convincingly.

What causes boils and abscesses?

Nearly all boils and skin abscesses begin the same undramatic way: a nick, a shaved hair, or a rubbed spot lets bacteria past the skin barrier, and the immune system responds by walling the invaders into a pocket. The organism is almost always Staphylococcus aureus, the most common pathogen involved in skin infections worldwide, regardless of age or geography. Its knack for producing severe skin disease traces to a handful of virulence factors rather than to sheer numbers.

The walling-off is a defense, and it is also the problem. Once pus is sealed inside a cavity with poor blood supply, neither immune cells nor antibiotics reach it well, which is why opening the pocket does what medication cannot. That logic runs through the entire care approach: incision and drainage is the recommended approach for carbuncles, abscesses, and large furuncles.

Bacteria that trigger them

Naming the organism is not academic; it changes which antibiotic would help if one is needed. Among adults arriving at US emergency departments with purulent skin infections, S. aureus was isolated in 76% of cases, and methicillin-resistant S. aureus accounted for 59% overall. More recent trial data puts MRSA lower but still substantial: it turned up in 45.3% of drained uncomplicated abscesses in one randomized trial and 49.4% of smaller abscesses in another.

Who is most at risk

Boils are ordinary, and the risk of getting one is not evenly spread. In UK primary care, first consultations for a boil or abscess ran 512 per 100,000 person-years in women and 387 in men, peaking between ages 16 and 34. A handful of factors shift those odds measurably:

None of these causes a boil on its own. They describe conditions that make it easier for a common organism to gain a foothold.

Join 150,000+ others building better health

Get the science behind better health, every week.

By clicking “Subscribe” you agree to our Terms of Service and Privacy Policy.

What do boils and abscesses look and feel like?

A boil announces itself as a red, tender, warm lump that grows over a few days and turns soft or springy in the middle as pus collects, often with a visible yellow-white head; clinically it is described as a painful, tender, fluctuant red nodule. Pressure and throbbing usually peak just before it points.

Some features suggest you are looking at more than one simple boil. Several openings, or a lump with multiple draining sinus tracts, points toward a carbuncle. Flat redness that keeps expanding outward with no soft center points instead toward cellulitis, whose defining feature is expanding erythema, warmth, tenderness, and swelling.

How a boil progresses, day by day

Timing varies from person to person, so read this as a sequence rather than a calendar. The underlying route is a folliculitis that deepens into a furuncle, and occasionally into a carbuncle.

  1. A tender red bump appears where a follicle was irritated, shaved, or rubbed.
  2. Over several days it enlarges and firms, and the skin around it reddens.
  3. The center softens as pus collects, sometimes forming a visible head.
  4. It either drains on its own or is drained by a clinician.
  5. The cavity closes, tenderness fades, and the redness settles last.

A lesion that keeps enlarging instead of settling has stopped following that arc, and it should be assessed.

Are boils and abscesses contagious?

The lump itself does not pass from person to person the way a cold does, but the bacteria behind it travel easily among people who share a home, a gym bench, or a razor. In one study of people with staph skin infections, S. aureus colonized 40% of index patients and 50% of their household contacts. Carriage is also easy to miss: swabbing the nose alone would have overlooked 48% of colonized people, because the organism also sits in the throat and groin.

Surfaces do the rest of the work. MRSA can survive on towels, razors, and furniture for hours to weeks and reach skin through cuts and scrapes. Household spread shows up in trial data too: after drainage, new infections among household members occurred in 1.7% of the antibiotic group versus 4.1% with placebo, which is evidence that these bacteria move between people, not a reason to seek antibiotics.

How to care for a small boil at home

Start with the rule that never bends: do not pick at or pop the bump or sore. Squeezing pushes bacteria sideways into healthy tissue and downward into the bloodstream, turning a contained problem into a spreading one. Home care has a narrower job than most people assume. Warmth and cleanliness encourage a small boil to come to a head and drain by itself, and that is all they are for. A boil that is large, deep, on the face, or simply not improving belongs with a clinician, not a washcloth.

The warm compress routine, step by step

Compress timing is common self-care practice rather than a number handed down by any guideline, so use these as sensible defaults.

  1. Wash your hands thoroughly before you touch the area.
  2. Hold a clean, warm, damp cloth against the lump for roughly 10 to 15 minutes, three or four times a day.
  3. Between compresses, keep it covered with a clean dry bandage.
  4. If pus drains, change the dressing and wash your hands again straight away.
  5. Launder the cloth, towels, and any bedding it touched rather than sharing towels, washcloths, or razors.

What not to do

Do: keep the lesion covered, wash your hands before and after touching it, launder towels and bedding that contact it, and use only your own razor.

Don't: squeeze or pop it, cut or drain it yourself, share towels, washcloths, or razors, sit in a shared tub or hot tub with an open lesion, or leave a draining lesion uncovered.

When a boil or abscess needs medical care

For a carbuncle, a true abscess, or a large boil, drainage is the definitive step, and no course of tablets substitutes for it. The judgment call is mostly about where the line sits.

SituationReasonable to manage at homeSee a clinician
SizeSmall, pea-sized, coming to a headLarger than a few centimeters, or still growing; a true abscess needs incision and drainage
LocationArm, leg, trunkFace, near the spine, or in the groin
Fever or feeling unwellNo systemic symptomsAny fever alongside the lesion
DurationImproving day by dayNo improvement within 48 hours, or still present after two weeks
Episodes per yearAn isolated lesionRepeat episodes; 10% of patients have another within 12 months
Diabetes or weakened immunityNot applicableAlways, since severely impaired host defenses change the calculus

Incision and drainage

The procedure is quicker and less dramatic than the name suggests. After local anaesthetic, the clinician opens the lesion, lets the pus out, and breaks up internal pockets with a curette or hemostat, often sending the material for culture. Packing the cavity afterward used to be automatic and is increasingly questioned: in a small randomized trial, packing did not reduce the need for a second procedure but produced significantly higher pain scores. Approaches differ between clinicians and settings, and abscess drainage is a defined emergency-department procedure. None of it is a do-it-yourself exercise.

When antibiotics are used

Whether to add an antibiotic after drainage is a clinician's call, weighed against the downsides. The decision turns on systemic signs of infection, severely impaired host defenses, multiple abscesses, extremes of age, or failure to respond to drainage alone. The evidence is honest about the size of the benefit. After drainage of an uncomplicated abscess, resolution reached 80.5% with an antibiotic versus 73.6% with placebo. For abscesses 5 cm or smaller, resolution at 10 days was 83.1% and 81.7% in the two antibiotic arms versus 68.9% with placebo, with more adverse events in the clindamycin arm. Most drained abscesses resolve without antibiotics; adding one improves the odds modestly and lowers the likelihood of spread.

Test 100+ biomarkers

One blood draw. A full picture of your health, explained in plain language.

Book your test

Red flags: when to see a doctor now

Certain features move a boil out of the wait-and-see category immediately.

Each of these is a reason to be seen, not a call to make at home.

How clinicians identify the cause

Most boils and abscesses are identified by looking and touching, and the honest caveat is that this is imperfect: clinical examination alone can be unreliable in distinguishing cellulitis from abscess. That uncertainty is exactly what bedside imaging resolves.

Across 14 studies and 2,656 patients, point-of-care ultrasound was 94.6% sensitive and 85.4% specific for abscess and corrected management in 10.3% of cases, sparing some people an unnecessary incision and catching pus in others whose skin looked merely inflamed. When a lesion is opened, the pus is often sent for culture, which is what tells a clinician whether MRSA is involved and which antibiotic would actually work if one becomes necessary.

Why boils keep coming back

Recurrence is common enough that a second boil should not feel like bad luck. Among UK primary-care patients who consulted for a boil or abscess, 10% had another within 12 months. US outpatient data runs higher, with 16.4% to 19.0% of culture-confirmed staph skin infections recurring within a year; among people without major comorbidities, most who recurred did so only once, and about half of those second episodes arrived within the first three months.

The usual mechanism is carriage. Some people simply live with S. aureus on their skin and in their nose, and nasal carriers face an increased risk of subsequent S. aureus infection. Clinicians sometimes respond with a decolonization regimen. In a German village where furunculosis kept circulating, nasal antibiotic ointment plus antiseptic washing ended transmission, and the same investigation found that contact with a case raised risk more than sixfold, which is why recurrence so often tracks households rather than individuals. Decolonization is not a universal fix, though; it reduces the risk of infection only in specific patient groups, making it a conversation to have with a clinician rather than a protocol to improvise.

Underlying conditions worth ruling out

Repeated boils can be the visible edge of something measurable underneath, and a pattern is reason enough to look. The most important look-alike is hidradenitis suppurativa, recognized by characteristic lesions in characteristic sites, meaning nodules, abscesses, tunnels, and scars in the armpits, under the breasts, groin, or perineum, with two or more flares in six months or lesions persisting three months or longer. It is uncommon rather than rare, affecting about 0.40% of people, and it is frequently missed for years. Blood sugar and body weight are the other two worth checking, since both diabetes and obesity track with higher skin-infection rates.

How to lower your risk of the next one

Lowering the risk comes down to two jobs: keeping the organism off broken skin, and reducing the conditions that let it settle in. The habits that do the first job are unglamorous and effective.

  • Wash your hands regularly and bathe after exercise
  • Keep cuts and scrapes clean and covered until they close
  • Do not share towels, washcloths, razors, or uniforms
  • Put a barrier, such as a towel or clothing, between bare skin and shared equipment or surfaces
  • Launder anything that touches a draining lesion

It helps to know where these infections concentrate: they cluster in spots where hair, clothing, or gear rubs the skin, which is why the neck, thighs, armpits, and buttocks come up so often.

The second job is slower and more personal. Weight, blood sugar, and smoking each track with a higher risk of a repeat boil, with obesity, diabetes, and smoking each carrying a relative risk of 1.3. They are associations, not verdicts, and they are the parts of the picture that can change.

How long boils and abscesses take to settle

Most small boils come to a head, drain, and settle within roughly one to three weeks, while a drained abscess usually starts improving within days of the pus being released. Trial data supports the shorter arc for drained lesions: among people with abscesses 5 cm or smaller, most participants had resolved at the 10-day assessment, including 68.9% of those who received placebo alone. Timeframes are typical, not guaranteed.

Slow recovery carries its own message. A lesion that has not improved within 48 hours of starting care, or that is still there after two weeks, calls for a clinician's assessment rather than more patience.

Check what's driving recurrent boils with a Superpower blood panel

One boil is a local problem. A pattern of them can reflect something systemic, and the usual suspects, blood sugar and metabolic status among them, happen to be measurable. They can also be present without a single symptom to give them away: diabetes still goes unrecognized in 9.5% to 17.8% of all diabetes cases in US adults. Superpower measures 100+ biomarkers. The Superpower Blood Panel covers glucose, hemoglobin A1c, high-sensitivity CRP, and white blood cell count, the metabolic and inflammatory markers that sit behind so much of how skin recovers, so the picture underneath is something you can actually see rather than guess at.

Frequently Asked Questions

References

  1. Stevens DL, Bisno AL, Chambers HF, et al. (2014). Practice guidelines for the diagnosis and management of skin and soft tissue infections: 2014 update by the Infectious Diseases Society of America. *Clinical Infectious Diseases*, *59*(2), e10–e52. https://doi.org/10.1093/cid/ciu444
  2. Troxell T & Hall CA (2023). *Carbuncle*. StatPearls Publishing. https://www.ncbi.nlm.nih.gov/books/NBK554459/
  3. Raff AB, Kroshinsky D (2016). Cellulitis: a review. *JAMA*, *316*(3), 325–337. https://doi.org/10.1001/jama.2016.8825
  4. Del Giudice P (2020). Skin infections caused by Staphylococcus aureus. *Acta Dermato-Venereologica*, *100*(9), adv00110. https://doi.org/10.2340/00015555-3466
  5. Tong SYC, Davis JS, Eichenberger E, Holland TL, Fowler VG Jr (2015). Staphylococcus aureus infections: epidemiology, pathophysiology, clinical manifestations, and management. *Clinical Microbiology Reviews*, *28*(3), 603–661. https://doi.org/10.1128/CMR.00134-14
  6. Moran GJ, Krishnadasan A, Gorwitz RJ, et al. (2006). Methicillin-resistant S. aureus infections among patients in the emergency department. *New England Journal of Medicine*, *355*(7), 666–674. https://doi.org/10.1056/NEJMoa055356
  7. Talan DA, Mower WR, Krishnadasan A, et al. (2016). Trimethoprim-sulfamethoxazole versus placebo for uncomplicated skin abscess. *New England Journal of Medicine*, *374*(9), 823–832. https://doi.org/10.1056/NEJMoa1507476
  8. Daum RS, Miller LG, Immergluck L, et al. (2017). A placebo-controlled trial of antibiotics for smaller skin abscesses. *New England Journal of Medicine*, *376*(26), 2545–2555. https://doi.org/10.1056/NEJMoa1607033
  9. Shallcross LJ, Hayward AC, Johnson AM, Petersen I (2015). Incidence and recurrence of boils and abscesses within the first year: a cohort study in UK primary care. *British Journal of General Practice*, *65*(639), e668–e676. https://doi.org/10.3399/bjgp15X686929
  10. Ghilotti F, Bellocco R, Ye W, Adami HO, Trolle Lagerros Y (2019). Obesity and risk of infections: results from men and women in the Swedish National March Cohort. *International Journal of Epidemiology*, *48*(6), 1783–1794. https://doi.org/10.1093/ije/dyz129
  11. Carey IM, Critchley JA, DeWilde S, Harris T, Hosking FJ, Cook DG (2018). Risk of infection in type 1 and type 2 diabetes compared with the general population: a matched cohort study. *Diabetes Care*, *41*(3), 513–521. https://doi.org/10.2337/dc17-2131
  12. Miller LG, Eells SJ, Taylor AR, et al. (2012). Staphylococcus aureus colonization among household contacts of patients with skin infections: risk factors, strain discordance, and complex ecology. *Clinical Infectious Diseases*, *54*(11), 1523–1535. https://doi.org/10.1093/cid/cis213
  13. Centers for Disease Control and Prevention (2025). *Preventing methicillin-resistant Staphylococcus aureus (MRSA)*. U.S. Department of Health and Human Services. https://www.cdc.gov/mrsa/prevention/index.html
  14. Centers for Disease Control and Prevention (2025). *Methicillin-resistant Staphylococcus aureus (MRSA) basics*. U.S. Department of Health and Human Services. https://www.cdc.gov/mrsa/about/index.html
  15. O'Malley GF, Dominici P, Giraldo P, et al. (2009). Routine packing of simple cutaneous abscesses is painful and probably unnecessary. *Academic Emergency Medicine*, *16*(5), 470–473. https://doi.org/10.1111/j.1553-2712.2009.00409.x
  16. Schmitz GR, Gottlieb M (2021). Managing a cutaneous abscess in the emergency department. *Annals of Emergency Medicine*, *78*(1), 44–48. https://doi.org/10.1016/j.annemergmed.2020.12.003
  17. Gottlieb M, Avila J, Chottiner M, Peksa GD (2020). Point-of-care ultrasonography for the diagnosis of skin and soft tissue abscesses: a systematic review and meta-analysis. *Annals of Emergency Medicine*, *76*(1), 67–77. https://doi.org/10.1016/j.annemergmed.2020.01.004
  18. Vella V, Galgani I, Polito L, et al. (2021). Staphylococcus aureus skin and soft tissue infection recurrence rates in outpatients: a retrospective database study at 3 US medical centers. *Clinical Infectious Diseases*, *73*(5), e1045–e1053. https://doi.org/10.1093/cid/ciaa1717
  19. Wertheim HFL, Melles DC, Vos MC, et al. (2005). The role of nasal carriage in Staphylococcus aureus infections. *Lancet Infectious Diseases*, *5*(12), 751–762. https://doi.org/10.1016/S1473-3099(05)70295-4
  20. Wiese-Posselt M, Heuck D, Draeger A, et al. (2007). Successful termination of a furunculosis outbreak due to lukS-lukF-positive, methicillin-susceptible Staphylococcus aureus in a German village by stringent decolonization, 2002-2005. *Clinical Infectious Diseases*, *44*(11), e88–e95. https://doi.org/10.1086/517503
  21. Saunte DML, Jemec GBE (2017). Hidradenitis suppurativa: advances in diagnosis and treatment. *JAMA*, *318*(20), 2019–2032. https://doi.org/10.1001/jama.2017.16691
  22. Jfri A, Nassim D, O'Brien E, Gulliver W, Nikolakis G, Zouboulis CC (2021). Prevalence of hidradenitis suppurativa: a systematic review and meta-regression analysis. *JAMA Dermatology*, *157*(8), 924–931. https://doi.org/10.1001/jamadermatol.2021.1677
  23. Fang M, Wang D, Coresh J, Selvin E (2022). Undiagnosed diabetes in U.S. adults: prevalence and trends. *Diabetes Care*, *45*(9), 1994–2002. https://doi.org/10.2337/dc22-0242

Led by doctors with 40 years of health and longevity expertise

Dr. Anant Vinjamoori

Dr. Anant Vinjamoori, MD

Chief Longevity Officer, Superpower

Dr. Leigh Erin Connealy

Dr. Leigh Erin Connealy, MD

Clinician & Founder of The Centre for New Medicine

Dr. Robert Lufkin

Dr. Robert Lufkin, MD

Physician & UCLA Medical School Professor, NYT bestselling author

Dr. Abe Malkin

Dr. Abe Malkin, MD

Founder & Medical Director of Concierge MD

Biological age 32 — a Superpower member portraitA health system built around you — test, understand, optimize100+ biomarkers across 2 blood drawsWhat to expect with Superpower — week 1 to month 3Trusted by 64,000+ customers — member reviewsBetter than the status quo — Superpower vs the old way60% of members found something a doctor missed — member survey
Daniel OSuperpower memberDavid LSuperpower memberRachel DSuperpower memberCamelia VSuperpower member

4.6 · 64,000+ members

Superpower health membership

Find early signs of 1,000+ conditions from a single blood draw - then act on what you find.

$199

/year*

100+ biomarkers from a single blood draw

Personalized plan that turns results into action

Superpower AI and on-demand care team

Results in 10 days or less

At-home or 2,000+ labs nationwide**

Yearly membership$0.55 / dayHSA/FSA eligible

*Pricing may vary for members in New York and New Jersey

**At-home labs for an additional cost

What we test

TestosteroneFree T4Vitamin DApoBHbA1cThyroid (TSH)hs-CRPFerritinCortisolFasting insulin

60%

found something a doctor missed

93%

rated it more useful than their checkup

77%

found something new in their results

Superpower member survey, 90 days post action plan (n=143).

How does the Superpower membership work?

We test 100+ biomarkers from a single blood draw each year. Your baseline test paints a comprehensive picture of your health, and your dashboard shows what’s working, what needs attention, and what to do next. Superpower AI and your care team help you take action across lifestyle, supplements, and treatments.

Can I cancel anytime?

Yes. You can cancel anytime, no fees. Your membership stays active through the end of your paid year.

How often should I test?

Your membership includes two blood tests per year: one to establish your baseline, and one to see whether your plan improved biomarkers that were out of range or not yet optimized. You can add other diagnostics anytime through the app or with help from your care team.

When will I get my results?

Book your draw at any of 2,000+ partner labs or at-home. Results are typically ready in about a week and land in the Superpower app.