Key Benefits

'- See how obesity impacts sugar, insulin, cholesterol, and inflammation right now.

• Spot early insulin resistance that drives weight gain and prediabetes.
• Flag rising glucose patterns that signal prediabetes or type 2 diabetes risk.
• Clarify fatigue, cravings, or crashes by linking them to sugar and insulin.
• Guide personalized nutrition, activity, and medication choices to improve heart and metabolic health.
• Protect your heart by identifying harmful cholesterol patterns and inflammation (hs-CRP).
• Support fertility and pregnancy by uncovering insulin resistance and inflammation linked to complications.
• Track progress from weight loss or therapy with repeat insulin, glucose, lipids, hs-CRP.

What are Obesity

Obesity biomarkers are measurable signals that show how your body manages energy, stores fat, and adapts to the strain of excess weight. They come from fat tissue, the pancreas, the liver, the gut, and the bloodstream, and together they reveal the biology beneath a rising scale number. Hormones that regulate hunger and fullness (ghrelin, leptin) and fat‑derived messengers (adipokines such as adiponectin) reflect appetite control and how responsive your cells are to insulin. Sugar‑ and insulin‑related markers (glucose, insulin, C‑peptide) capture insulin resistance, a core driver of metabolic stress. Blood fats (triglycerides, HDL) and liver signals (ALT, GGT) show how fat is being packaged, burned, or stored—especially in the liver. Inflammation and metabolic stress markers (CRP, uric acid) indicate the low‑grade inflammation that often accompanies excess adiposity. Testing this mix creates a metabolic profile that distinguishes different “flavors” of obesity—dominated by insulin resistance, appetite signaling, fatty liver, or inflammation—and helps tailor nutrition, activity, sleep, and medications. Repeating tests over time tracks whether your plan is repairing the underlying biology, not just changing weight.

Why are Obesity biomarkers important?

Obesity biomarkers are the measurable signals of how the body is handling energy, storing fat, and mounting inflammation. Together—insulin, glucose, lipids, and hs‑CRP—they map the crosstalk between pancreas, liver, muscle, fat tissue, blood vessels, and the immune system, revealing risk before disease is obvious.

Within reference ranges, patterns matter. Fasting insulin is most reassuring at the low end of normal, reflecting good insulin sensitivity. Fasting glucose is healthiest in the lower-to-mid normal range, suggesting stable hepatic glucose control. In a standard lipid panel, LDL cholesterol is best toward the low end, HDL toward the higher end, and triglycerides toward the low end; this profile signals efficient lipid transport and less atherogenic particle burden. hs‑CRP is most favorable near the bottom of its range when no infection is present, indicating quiet systemic inflammation.

When values are unusually low, they tell their own story. Very low insulin with low glucose can point to undernutrition or impaired pancreatic output; symptoms may include shakiness, sweating, or confusion from hypoglycemia. Very low glucose suggests hypoglycemia, more likely in children, small-bodied adults, or those with medication-related risk. Markedly low LDL or triglycerides may reflect malabsorption or hyperthyroidism, though mildly low triglycerides are often benign. A very low hs‑CRP simply indicates minimal inflammation. Women typically have higher HDL than men; pregnancy physiologically raises insulin and lipids; puberty can transiently lower insulin sensitivity.

Big picture: these biomarkers tie adipose biology to cardiovascular, metabolic, and immune health. Tracked over time, they forecast risks for diabetes, fatty liver, heart disease, and reproductive and sleep disorders, turning diffuse physiology into actionable early warning.

What Insights Will I Get?

Obesity reflects how the body regulates energy storage, fuel use, and inflammation across organs, shaping cardiovascular, cognitive, reproductive, and immune health. At Superpower, we test these specific biomarkers: Insulin, Glucose, Lipids, hs-CRP.

Insulin, the pancreatic hormone that drives glucose uptake and fat storage, often runs higher in obesity due to insulin resistance. Glucose, the circulating fuel, rises when hepatic output and peripheral disposal are impaired. Lipids (triglycerides, HDL, LDL) show transport balance; obesity favors high triglycerides, low HDL, and small dense LDL. hs-CRP is a high-sensitivity measure of systemic inflammation, elevated by inflamed or enlarged adipose tissue.

Healthy stability looks like fasting insulin in a low, steady range with proportionate meal responses, signaling metabolic flexibility. Glucose that stays in normal fasting range with modest variability reflects effective liver and muscle glucose handling. Lipids with lower triglycerides, higher HDL, and LDL in recommended ranges indicate efficient lipid metabolism and lower atherogenic burden. Low hs-CRP indicates quiet chronic inflammation, healthier adipose signaling, and steadier vascular function.

Notes: Results vary with fasting status, acute infection or injury, strenuous exercise, pregnancy, age, sleep and stress, and menstrual phase. Medications (e.g., steroids, antipsychotics, beta-agonists, statins, metformin) and assay differences or genetic dyslipidemias can shift values.